By Aaron Hermann
A couple of nights ago Bob and I were discussing the list of diseases making headlines and how they might be related to the bioforming pandemic we have been watching for. Part of our discussion focused on the overwhelming amount of signaling that has been taking place pertaining to a possible pandemic. "They" have literally signaled everything from A-to-Z, which will make it nearly impossible to trace anything back to the parties responsible in the event a bioweapon is released.
I believe it is important to continue pointing out the various scenarios being signaled, and relaying what it seems the Lord is continuing to bring forth as what is going on really is the world's biggest deal. I believe the Lord granted me some insight into what may be occurring with these seemingly unrelated diseases during my conversation with Bob.
It may be that the record-breaking number of cases of West Nile Virus, the H3N2 Swine Flu which has demonstrated the ability for human-to-human transmission, the parallels of the Swine-origin H1N2 containing a gene from the 2009 H1N1 which further increases the likelihood of human-to-human transmission, and the Hantavirus which often causes fatal pulmonary complications may be separate components of a government funded bioweapon released in various installments that will reassemble as the diseases converge in nature.
It seems that this particular scenario is rather plausible, especially in light of the fact that in addition to the coverage of the West Nile Virus outbreak and the variant forms of flu the Hantavirus is now beginning to receive significant attention as it is being presented as a global threat. Even the mainstream news is covering the current outbreak of the severe respiratory disease that supposedly originated in Yosemite Park, which poses a threat to 10,000 people from around the world who visited the park between June and July of this year.
I find it rather telling that the incubation period of the disease is generally six weeks, and the target time frame of exposure just happens to coincide with the same time-frame of the 2012 London Olympic Games. Could this be yet another example of the "sleight of hand" used by the shill media to lead attention away from the fact that perhaps there is a connection to the Olympics? Perhaps. Bob and I wrote about this scenario in Part 24 - 2012 London Olympics - Our Goose Is Cooked! and Part 26 - 2012 London Olympics - Our Goose is Cooked: Will That Be Take Out or Delivery?
In its natural form the Hantavirus is a very serious disease and allowing for the very real possibility of man-made mutations and viral engineering having taken place this could become a very serious global issue.
“in Nombre hantavirus is the cause of most hantavirus cases in the United States and Canada; deer mice carry this form, the CDC reported. There are also less common forms of hantavirus, such as the New York hantavirus, which is carried by the white-footed mouse, and the Black Creek hantavirus, which is carried by the cotton rat.
Hantavirus leads to hantavirus pulmonary syndrome. The condition starts out with flu-like symptoms -- like vomiting, muscle aches and fever -- but then can progress to serious symptoms like fluid in the lungs, cough with secretions, decreased efficiency of the heart and decreased blood pressure, the Mayo Clinic reported.
If the symptoms get bad enough, it can lead to lung failure, according to the A.D.A.M. Medical Encyclopedia. In fact, if the disease has reached the lungs, the prognosis is not good even among people who get treatment.
The CDC reported that 38 percent of people who contract hantavirus die.”
Hantavirus: What Are The Symptoms Of The Virus That Killed 2 At Yosemite Camp?
Hantavirus leads to hantavirus pulmonary syndrome. The condition starts out with flu-like symptoms -- like vomiting, muscle aches and fever -- but then can progress to serious symptoms like fluid in the lungs, cough with secretions, decreased efficiency of the heart and decreased blood pressure, the Mayo Clinic reported.
If the symptoms get bad enough, it can lead to lung failure, according to the A.D.A.M. Medical Encyclopedia. In fact, if the disease has reached the lungs, the prognosis is not good even among people who get treatment.
The CDC reported that 38 percent of people who contract hantavirus die.”
Hantavirus: What Are The Symptoms Of The Virus That Killed 2 At Yosemite Camp?
I have included these links in the event you are interested in learning more about Hantavirus:
- CDC says 10,000 at risk of hantavirus in Yosemite outbreak
- Yosemite expands hantavirus warning
- Another Yosemite camper dies in hantavirus outbreak
One of the primary distinguishing characteristics that determines the lethality of a respiratory illness is how deeply the infection penetrates the lungs. Seasonal flu remains primarily in the mouth, throat, and trachea portions of the body which makes it quite contagious, but not very lethal. The bird flu on the other hand deeply penetrates the lungs, which makes it very deadly but not contagious...at least not yet! In Part 2 - A Bioforming Pandemic: U.S. Government Funded Killer Bird Flu Research ~ Preparing for Global "9-11" Event? I wrote at length about the U.S. Government funded research done by Ron Fouchier which mutated the bird flu making it both lethal and contagious.Earlier this year scientists were able to identify the presence of a gene that aids the body in protecting the lungs from the presence of infection. The presence of the "IFITM 3" gene proves to be a significant factor determining the severity of symptoms and prognosis of someone suffering from a respiratory infection.
“A genetic discovery could help explain why flu makes some people seriously ill or kills them, while others seem able to bat it away with little more than a few aches, coughs and sneezes.
In a study published in the journal Nature on Sunday, British and American researchers said they had found for the first time a human gene that influences how people respond to flu infections, making some people more susceptible than others.
The finding helps explain why during the 2009/2010 pandemic of H1N1 or "swine flu", the vast majority of people infected had only mild symptoms, while others - many of them healthy young adults - got seriously ill and died.
In future, the genetic discovery could help doctors screen patients to identify those more likely to be brought down by flu, allowing them to be selected for priority vaccination or preventative treatment during outbreaks, the researchers said. It could also help develop new vaccines or medicines against potentially more dangerous viruses such as bird flu.
Paul Kellam of Britain's Sanger Institute, who co-led the study and presented the findings in a telephone briefing, said the gene, called ITFITM3, appeared to be a "crucial first line of defense" against flu. When IFITM3 was present in large quantities, the spread of the virus in lungs was hindered, he explained. But when IFITM3 levels were lower, the virus could replicate and spread more easily, causing more severe symptoms.
People who carried a particular variant of IFTIM3 were far more likely to be taken into hospital when they got flu than people who carried other variants, he added. "Our research is important for people who have this variant as we predict their immune defenses could be weakened to some virus infections," Kellam said.
"Ultimately as we learn more about the genetics of susceptibility to viruses, then people can take informed precautions, such as vaccination to prevent infection."
MICE EXPERIMENTS HELPED MAKE BREAKTHROUGH
The potential antiviral role of IFITM3 in humans was first suggested in studies conducted by Abraham Brass of the Ragon Institute and Gastrointestinal Unit of Massachusetts General Hospital in the United States. Using genetic screening, he found that it blocked the growth of flu and other viruses in cells.
Teams led by Brass and Kellam then took the work further by knocking out the IFITM3 gene in mice. They found that once these animals contracted flu they had far more severe symptoms than mice with the IFITM3 gene.
In effect, they said, the loss of this single gene in mice can turn a mild case of influenza into a fatal infection. The researchers then sequenced the IFITM3 genes of 53 patients who had been hospitalized with seasonal or pandemic flu and found that a higher number of them had a particular variant of IFITM3 compared to the general patient population.
The researchers believe this variant results in a shorter version of the protein or one that is less abundant in cells, leaving patients more vulnerable to flu when they get it.
"Our efforts suggest that individuals and populations with less IFITM3 activity may be at increased risk during a pandemic, and that IFITM3 could be vital for defending human populations against other viruses such as avian influenza," said Brass.”
Scientists find gene that can make flu a killer
In a study published in the journal Nature on Sunday, British and American researchers said they had found for the first time a human gene that influences how people respond to flu infections, making some people more susceptible than others.
The finding helps explain why during the 2009/2010 pandemic of H1N1 or "swine flu", the vast majority of people infected had only mild symptoms, while others - many of them healthy young adults - got seriously ill and died.
In future, the genetic discovery could help doctors screen patients to identify those more likely to be brought down by flu, allowing them to be selected for priority vaccination or preventative treatment during outbreaks, the researchers said. It could also help develop new vaccines or medicines against potentially more dangerous viruses such as bird flu.
Paul Kellam of Britain's Sanger Institute, who co-led the study and presented the findings in a telephone briefing, said the gene, called ITFITM3, appeared to be a "crucial first line of defense" against flu. When IFITM3 was present in large quantities, the spread of the virus in lungs was hindered, he explained. But when IFITM3 levels were lower, the virus could replicate and spread more easily, causing more severe symptoms.
People who carried a particular variant of IFTIM3 were far more likely to be taken into hospital when they got flu than people who carried other variants, he added. "Our research is important for people who have this variant as we predict their immune defenses could be weakened to some virus infections," Kellam said.
"Ultimately as we learn more about the genetics of susceptibility to viruses, then people can take informed precautions, such as vaccination to prevent infection."
MICE EXPERIMENTS HELPED MAKE BREAKTHROUGH
The potential antiviral role of IFITM3 in humans was first suggested in studies conducted by Abraham Brass of the Ragon Institute and Gastrointestinal Unit of Massachusetts General Hospital in the United States. Using genetic screening, he found that it blocked the growth of flu and other viruses in cells.
Teams led by Brass and Kellam then took the work further by knocking out the IFITM3 gene in mice. They found that once these animals contracted flu they had far more severe symptoms than mice with the IFITM3 gene.
In effect, they said, the loss of this single gene in mice can turn a mild case of influenza into a fatal infection. The researchers then sequenced the IFITM3 genes of 53 patients who had been hospitalized with seasonal or pandemic flu and found that a higher number of them had a particular variant of IFITM3 compared to the general patient population.
The researchers believe this variant results in a shorter version of the protein or one that is less abundant in cells, leaving patients more vulnerable to flu when they get it.
"Our efforts suggest that individuals and populations with less IFITM3 activity may be at increased risk during a pandemic, and that IFITM3 could be vital for defending human populations against other viruses such as avian influenza," said Brass.”
Scientists find gene that can make flu a killer
While the information was presented in such a way as to make the ignorant and unsuspecting believe the research was being done for the benefit of mankind, the title of the article tells a much different story. Is it me, or does the title of "Scientists find gene that can make flu a killer" seem just a bit ominous?
I have included the following links if you are interested in reading more about the connection between the IFITM3 gene and the lethality of the flu, and other respiratory infections.
- Gene Explains Why Flu Can Be Serious
- Genetics of Flu Susceptibility: Why the Flu Is Life-Threatening for Some, and Quite Mild for Others
- IFITM3 Variant Linked with Worse Flu Symptoms, Need for Hospitalization
With the dense amount of information presented thus far you may have missed the significance of "knockout mice" and what that may mean for the global population. By definition a "knockout mouse" is:
“A knockout mouse is a genetically engineered mouse in which researchers have inactivated, or "knocked out", an existing gene by replacing it or disrupting it with an artificial piece of DNA. The loss of gene activity often causes changes in a mouse's phenotype, which includes appearance, behavior and other observable physical and biochemical characteristics.”
Wikipedia - Knockout Mouse
Wikipedia - Knockout Mouse
If scientists have figured out how to identify and "knock out" genes in mice, I believe that we can rather safely assume that this type of activity can also be done to humans. Several articles have appeared recently touting the scientific advancement in the field of genetics and DNA. To assume that these articles represent the latest advancements in the field would be naive. I think it is safe to say that what is currently being reported is just the "tip of the iceberg" of what is presently being done in these fields.
With these advancements in understanding how genes and DNA work, it is reasonable to assume they have learned how to manipulate our DNA, switch genes on and off, and further alter God's creation of Adamic-Mankind.
If "they" have figured out a way to "knock out" the IFITM3 gene in those they wish to kill with their bioweapon, then I am convinced that they also have designed this bioweapon as a means of enhancing those with certain genetic characteristics through this viral loading procedure, which was covered in Part 1 - A Bioforming Pandemic: Killing Some and Enhancing Others.
When you read or hear the official reports of outbreaks and there's a nagging sense that there's something they're not telling us, that's probably the holy spirit. The big picture evidence suggests that the official stories are bogus, contrived, that we are being lied to. Strings are being pulled behind the scenes directing the outcome of events. There really is a conspiracy against us. In the face of the onslaught of lies and deceit hold fast to what the Lord has given you and what the Holy Spirit has revealed to you as true.
In the face of all the overwhelming physical evidence to the contrary, my Lord has this entire situation under control and only that which He has ordained will be allowed to come to pass. Hang in there folks, soon all of our troubles will indeed be over!
More to come...Lord willing!
By Aaron and Bob
Bob Schlenker (theopenscroll@) and Aaron Hermann (theultimateplan@)
Bob,
ReplyDeleteLooks like another big warning.
http://www.bbc.co.uk/news/world-europe-19555534